Harada syndrome and its ophthalmologic repercussions: a case report
Abstract
Also known as uveomeningoencephalic syndrome, its ocular involvement is marked by severe bilateral panuveitis associated with serous retinal detachment (Mota et al, 2010). SVKH is divided into four distinct phases: prodromic, which precedes ocular inflammation by 3 to 5 days, extends for 1 to 2 weeks,(2, 3) mimics a viral infection, and presents as characteristics: fever, headache, orbital pain, photophobia, tinnitus, meningismus and liquoric pleocytosis; uveitic (acute), which begins 3 to 5 days after the prodromic phase and lasts weeks to months, in which there is diffuse inflammation of the choroid, responsible for the subsequent accumulation of subretinal fluid, characteristic of this phase of the disease, and variable extension of the inflammation, usually granulomatous, to the anterior segment; the convalescent phase follows gradually with regression of inflammation and depigmentation of the uvea and skin; this quiescent stage is interrupted in 17 to 73% of cases by the chronic phase of the disease, presenting mostly as recurrent anterior uveitis (Costa et al, 2018)
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